What´s Lactoferrin?

Lactoferrin is an iron-binding glycoprotein that exists in high concentrations in breast milk and other external secretions in the body. In healthy individuals, normal serum levels range from 2-7 μg / ml, but concentrations can be higher during inflammation and sepsis (Cornish et al., 2004). Since lactoferrin has the ability to retain iron in a wide pH range, it is involved in the iron homeostasis and acts as a multiple effect factor (Amini & Nair, 2011). All vertebrate species have an iron restraint defense system to remove and isolate excess iron from the body.

A discussion about lactoferrin is that the iron level increase we see (roughly the same increase as ferrous sulfate) is due to the ability of lactoferrin to administer iron or to reduce inflammation. It seems to be more related to its antimicrobial properties, and it is generally not recommended to use it for iron deficiency, but it may be used on other irons. Since most lactoferrin supplements are in the form of depleted iron, if it can be obtained with a combination of iron, it may increase iron, but more research is needed. For those who are sensitive to iron overload, it is worth investigating

Lactoferrin is an iron-containing glycoprotein secreted from most mucous membranes. It is the main component of the secondary polymorphonucleus, the main cell of the acute inflammatory response. In intestinal inflammation, white blood cells invade the mucosa and excretion of lactoferrin into feces increases. In addition to calprotectin, fecal lactoferrin is clearly a biomarker most widely used for intestinal inflammation. Frequency of drug-induced colitis is usually underestimated. Studies have shown that colitis can be caused by antibiotics, NSAIDs, laxatives, vasoconstrictors, and estrogenic progesterone. Pseudomembranous colitis is caused by C. It is caused by antibiotics that promote excessive growth of difficile. Antibiotics such as penicillin, amoxicillin, ampicillin, etc. may cause hemorrhagic colitis. Ischemic colitis may be caused by drugs inducing mesenteric vasoconstriction