The Relationship Between and Effects of Adolescents Electronic Media Use and Delayed Sleep Phase Disorder

The relationship between the use of electronic media and delayed sleep stage disorder and the effects on children and adolescents is very difficult at school because of the delayed sleep disorder (DSPD) caused by electronic media in the bedroom. It can be explained as a period of rest of the body and the mind, during which the will and consciousness are partially or completely interrupted, and the body function is partially interrupted.

There are two main sleep schedule barriers: delayed sleep and delayed sleep. In the former, the start and transition of sleep occurs earlier than social norms, the latter comes to be delayed in sleep, and happens late in the later days than expected. These changes in the sleep-wake cycle can occur after traveling abroad or overseas traveling beyond time zones. They can also occur over long periods without obvious environmental factors. Various genes involved in the regulation of circadian rhythms have been elucidated and show the presence of genetic elements in specific examples of sleep schedule disorders. These symptoms can be treated by gradually adjusting the sleeping time. This readjustment can be promoted by physical (eg light irradiation) and pharmacological (eg melatonin).

Aging is related to changes in the relationship between sleep time and circadian rhythm, and the rhythm is slower during sleep than young adults. In order to investigate whether the difference in phase delay response to light in the evening contributed to this, we conducted a nine-day hospitalization test for 10 healthy elderly (over 65 years old) subjects. We evaluated the circadian rhythm phase with routine procedures and reevaluated the circadian rhythm phase, exposing each subject to a 5 hour broad spectrum light stimulus from the early biological night. The stimulation ranges from very dark (~ 2 lux) to very bright (~ 8,000 lux) indoor lights. We found a significant dose-response relationship between illumination and phase shift of melatonin rhythm; evidence suggests that sensitivity is different from sensitivity in young adults rather than maximum response to light. These findings indicate that phase delay due to aging reduces response to moderate light levels.