Pesticide Exposures

The term insecticide refers to insecticides, herbicides, fungicides, disinfectants, and various other substances used to kill, repel or control the life of any plant or animal considered a pest, . Pests occur in places where they are not needed or are creatures or organisms that harm human or other animals.

Pesticides can bring various benefits if used properly. They can increase crop yield, protect agricultural products, prevent the spread of insects, and control alien species. However, pesticides may also cause harm. About one billion pounds of pesticide active ingredient is used every year in the United States, and there are over 20,000 pesticide products in the market. In 2012, pesticides were the tenth leading cause of addiction exposure reported by the American Addiction Control Center.

People may come in contact with pesticides used in homes, schools, hospitals and workplaces. Many well-known household items are insecticides such as insect repellents, detergents, herbicides, etc. With the widespread use of pesticides in food production people are exposed to low levels of pesticide through residues in food and contaminated drinking water. Scientists are not yet clearly understanding the impact of pesticide exposure on chronic health

The US Environmental Protection Agency (EPA) is in charge of approval of pesticides and assessment of pesticide harm to humans.

Call a person who has been exposed to pesticides for more than 10 hours after mailing the questionnaire; mixed pesticide exposure (including herbicides, insects, fungi and fumigants); pesticides reported by case and control Classified exposure chemicals, organophosphorus pesticides, dicamba (OR 1.38 - 1.92, statistical properties) increased the number of days / years of NHL risk by adjusting several covariates, still OR 1.32 - 2.33 and stat. For 2,4-D, Mecoprop, Dicamba, several insecticides; OR adjusted for covariates more than 1.96 - 3.42 in statistical data. Meaning Dicamba, Mecoprop, Aldrin

Children are mainly studying the respiratory effects of exposure to pesticides in the environment. In our study, 14 studies on children have been confirmed, ie 7 parenteral pesticide exposure assessments at different age postpartum, and 7 pesticide exposure assessments. In eight studies, pesticides or metabolites in body fluids were detected in maternal blood (n = 3) or cord blood (n = 2) of child's blood (n = 1) ). 2). In other studies on children, exposure was assessed indirectly through questionnaires.

Six studies examined adult radiation exposure. One study used biomarkers to assess pesticide exposure, three studies evaluated the proximity of the house to the field or spray area, and one study evaluated pesticide exposure through a questionnaire did. Four studies were conducted in Spain to evaluate the relationship between pre-natal organic chlorine pesticide exposure and pediatric asthma. Sunyer et al. Between 1997 and 1998, 482 mothers were followed for 12 months in Menorca during prenatal care and 405 children were followed up until 6 years old. The authors found that the concentration of dichlorodiphenyl dichloroethylene (DDE), a metabolite of dichlorodiphenyl trichlorethylene (DDT) in cord blood at birth, was associated with an increased risk of wheezing at 4 years of age ( Relative risk is 2.36, 95% CI 1.19 - 4.69) highest quartile (> 1.90 ng · mL -1)