Pathological Book Review on Multiple Sclerosis

Many people around the world are suffering from nervous weakness such as multiple sclerosis. Multiple sclerosis is best explained as a pathological "inflammatory demyelinating disease in the human central nervous system" and has over 5 million people worldwide (Trapp & Nave, 2008). As with other chronic diseases, in dealing with multiple sclerosis, many people choose to write down their experience, including the overall fight against the way they get diagnosed and the disease.

Three diseases fall into this category: multiple sclerosis, acute disseminated encephalomyelitis (ADEM) and acute hemorrhagic leukoencephalitis (AHL). The most common of these multiple sclerosis is pathological and pathological heterogeneity, based on and based on clinical and pathological features and based on four major subtypes , Acute, optic neuromyelitis and concentric sclerosis). We will further fragment the type of plaque. Combination of morphological and immunohistochemical results

Multiple sclerosis is a disease affecting the central nervous system. Sclerosis refers to scar tissue, and therefore multiple sclerosis is multiple scar tissue on the central nervous system. One in 1,600 people was diagnosed with multiple sclerosis. In the United States, about 350,000 people suffer from multiple sclerosis. It is incurable, but there are still a lot of drugs and lots of research to help fight disease. The cause of multiple sclerosis is unknown, but there are many theories. Some researchers believe that it may be a virus or immune system imbalance. Others think that it may be an environmental problem. Researchers believe that it may be an environmental factor as there are few people (if any) who live near the equator. Multiple sclerosis usually occurs in women between 30 and 50 years old. Multiple sclerosis is also common in black people and Asians. This disease is fatal, infectious, or not directly inherited.

The pathological similarity of multiple sclerosis and experimental allergic encephalomyelitis and detection of T cells reactive with myelin components has been explained as evidence that multiple sclerosis is an autoimmune disease 9. As a contrary evidence, other investigators point out that they can not induce demyelination due to patient serum or T cell metastasis, and that there are no immunological markers specific for most disease forms. In the role of multiple sclerosis the most common are Epstein-Barr virus and Human Herpesvirus 6.11, 12. Possible mechanisms include direct viral injury to the central nervous system, immune disorders leading to immunogenic exposure of the myelin antigen and subsequent myelin itself, as well as molecular mimics between the viral antigen and myelin Contribute