Memory, Aging, and the I-function

The folk wisdom of memory, aging, and I function long recognized the memory capacity declining as individuals aged. Wrinkled skin. The effect of time on the human brain is more complicated than simple progressive reduction until one washes this deadly coil. The effects of memory loss and cognitive decline do not affect how we understand how memory works in treating debilitating diseases such as Alzheimer's disease and 'I function'. Learning and memories

The aging process usually results in changes in brain function and function, causing problems such as loss of memory and mental deterioration. Age is a major risk factor for the most common neurodegenerative disorders, including mild cognitive impairment, Alzheimer's disease, cerebrovascular disease, Parkinson's disease, and Rougher's disease. A lot of research has focused on aging diseases, but there are only a few information studies on molecular biology of the aged brain. Much of the molecular change is due to the reduction in the size of the brain and the loss of brain plasticity. Brain plasticity is the ability of the brain to change structure and function. The main function of the brain is to decide what information is worth holding and which information is not worth it; if there are actions and ideas that no one uses, the brain eliminates the space I will.

Because of the aging of society, cognitive functions increase the threat of advanced cognitive function, so it is essential to understand molecular and cellular events that control brain memory and learning processes. Here we show that a new major gene pair - | - SRGAP 2 - FAM 72 - | (SLIT - ROBO Rho GTPase activating protein 2, a family similar to 72 sequence) is involved in the regulation of neural stem cell (NSC) gene expression Reveal a new doctrine. It is a unique participant in controlling the plasticity of the human brain. Insight into the specific regulation of brain specific neuroblast genes | - SGGAP2 - FAM 72 - | New treatment that can fundamentally help in the recovery or improvement of higher cognitive function

Brain plasticity, cognitive function and neural stem cells: important role of brain-specific neurogenic genes | - SGGAP 2 - FAM 72 - |

Amnestic MCI is characterized by selective damage to episodic memory function exceeding expectation for age. Episodic memory impairment is also a primary feature of normal senescence associated with recovery strategies and is the earliest and most serious cognitive impairment in Alzheimer's disease, but mainly involves coding (Carlesimo et al., 1998; Deweer et al. , 2001). . Interestingly, it is unknown which aspects of episodic memory are most vulnerable to MCI attacks. Similar cognitive strength and free recall damage suggests that coding defects are dominant and recovery problems may not be the main cause of episodic memory impairment at this stage (Almkvist et al., 1998). Recent research on Wang and Zhou (2002) further strengthened this perspective. They used experimental procedures based on previous functional imaging studies of normal subjects to show that coding and searching processes involve different nervous systems (Gabrieli et al., 1997).