Exploring Eye Motion and Models of Head Centered Motion Perception

4: Head center motion perception model combining incoming signal and outgoing signal. In order to maintain a stable and accurate world view and to estimate the external motion, we need information on our body movement and sensory signals on the movement of external objects. Spatial homeostasis is a vision system that maintains the vision of the outside world and does not jump or move with eye movement (Deubel, Bridgeman, & Schneider, 1998; Stark & ​​Bridgeman, 1983).

It is meaningful to pursue the relationship between eye movement and motion perception. During tracking, the fMRI signal associated with motor perception is enhanced at V 5 and later posterior occipital area (Barton et al., 1996 b; Freitag et al., 1998). Some of the neural activity in the tracked area V 5 a may be a reference copy on the eye movements themselves. Since you can cause movement of the image on the retina by eye movements while moving the object while the eye is stationary or while the world is stationary, you can use both duplication of the video . A subject with a vertigo caused by a moving object can not take his own eye movement into account when estimating the motion of that object (Haarmeier et al., 1997). He has bilateral occipital lesions, it may be a homolog of V5a.

Are different types of motion perception affected by different lesions? Studies have shown that there can be a significant difference between the subject and the pattern of the affected exercise process (Vaina et al., 2005). Primary motion refers to the calculation of the motion stimulus by correlating the spatial distribution of the luminance in the visual scene with the passage of time. However, in addition to brightness, movement can be distinguished from other information such as contrast, texture, stereoscopic viewing, flickering. These are called secondary motions. In the first case study, the first and second order movements may have different trajectories, and the second order motion is influenced by the lesion near the V5 region (Vaina and Cowey, 1996; Vaina et al., 1999) , The primary motion affects V2 and V3. Inner occipital lesion (Vaina et al., 1998), however recent research found that primary and secondary motor cognitive deficits coexist with the V 5 region (Greenlee and Smith, 1997; Braun et al., 1998) .