Cerebellar Lesions

Cerebellar lesions According to the article by Dr. Rhawn Joseph, each deep cerebellar nucleus maintains a semi-independent human figure (2000). It is estimated that the cerebellum is active and has a stabilizing effect on motor function; therefore, in addition to affecting time, it is also functionally responsible for coordinated smooth motor fine-tuning is there. Not only is the cerebellum associated with motor function, but importantly, it is also related to classical regulation: learning a new exercise program (Joseph, 2000), for example.

The reason for language impairment in patients with cerebellar lesions is that the cortex can pay attention to only one thing at a time. While the cerebellar function is busy with a conscious brain concentrating on thinking, conversation, or other things, it is a routine and important task such as standing up and rising, walking, maintaining balance and posture Is to process. Without the help of the cerebellum, the new cortex is forced to consciously perform daily tasks. In other words, the new cortex can not be balanced, posture, or upright at the same time. This is the reason we need the cerebellum.

Ancient inexplicable facts about brains unknown to neuroscientists: the cerebellum does not produce speech

Many patients with cerebellar injuries learn to respond by quickly switching tasks within 1 second. This allows you to pause and talk when sitting or standing. According to the lesions their body often shivers when they speak. But as they are lying down, their voices will return to normal. They no longer need to consciously maintain posture or balance, so they can talk freely without interference. If the cause of the language disorder is the cerebellum, this will not happen. The problem of utterance occurring after cerebellar lesion is obviously a side effect, not a cerebellar language production capacity.

Ancient inexplicable facts about brains unknown to neuroscientists: the cerebellum does not produce speech

Damage to nerve cells may cause damage to the spinal cord ganglia; they may also occur in reticularoids, vestibular nuclei, cerebellar sites, and deep cerebellar nuclei. Inflammation associated with the destruction of nerve cells usually changes the color and appearance of the gray matter of the spine and shows reddish swelling. Other destructive changes associated with paralytic diseases occur in forebrain areas, particularly in the hypothalamus and thalamus. The molecular mechanism of poliovirus causing paralytic disease is unknown